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Chymase uptake by cardiomyocytes results in myosin degradation in cardiac volume overload.

Heliyon. 2019; 
PowellPamela C，WeiChih-Chang，FuLianwu，PatBetty，BradleyWayne E，CollawnJames F，Dell'ItaliaLou
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Custom Vector Construction … The rMcpt2 cDNA flanked by the BamHI and NotI restriction sites (sequence was shown below) were synthesized and cloned to the pBluescript II KS(+) vector through the service provided by GenScript (Piscataway, NJ, USA) … Get A Quote

摘要

Volume overload (VO) of isolated mitral regurgitation (MR) or aortocaval fistula (ACF) is associated with extracellular matrix degradation and cardiomyocyte myofibrillar and desmin breakdown. Left ventricular (LV) chymase activity is increased in VO and recent studies demonstrate chymase presence within cardiomyocytes. Here we test the hypothesis that chymase within the cardiomyocyte coincides with myosin and desmin breakdown in VO.

关键词

Cardiology，Cell biology，Molecular bio
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